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| Porcine Knock-in Fibroblasts Expressing hDAF on α-1,3-Galactosyltransferase (GGTA1) Gene Locus |
| Ji Woo Kim, Hye-Min Kim, Sang Mi Lee, Man-Jong Kang |
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Anim Biosci. 2012;25(10):1473-1480. Published online August 22, 2012 DOI: https://doi.org/10.5713/ajas.2012.12146 |
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Porcine Knock-in Fibroblasts Expressing hDAF on α-1,3-Galactosyltransferase (GGTA1) Gene Locus Asian-Australasian Journal of Animal Sciences. 2012;25(10):1473-1480  Allelic variation of the porcine α-1,3-galactosyltransferase 1 (GGTA1) gene Journal of Applied Genetics. 2008;49(1):75-79 Production of heterozygous alpha 1,3-galactosyltransferase (GGTA1) knock-out transgenic miniature pigs expressing human CD39 Transgenic Research. 2016;26(2):209-224 Optimization of a CRISPR/Cas9-mediated Knock-in Strategy at the Porcine Rosa26 Locus in Porcine Foetal Fibroblasts Scientific Reports. 2017;7(1): Significance of the Evolutionary α1,3-Galactosyltransferase (GGTA1) Gene Inactivation in Preventing Extinction of Apes and Old World Monkeys Journal of Molecular Evolution. 2014;80(1):1-9 Efficient Knock-in of a Point Mutation in Porcine Fibroblasts Using the CRISPR/Cas9-GMNN Fusion Gene Genes. 2018;9(6):296 The combinational use of CRISPR/Cas9-based gene editing and targeted toxin technology enables efficient biallelic knockout of the α-1,3-galactosyltransferase gene in porcine embryonic fibroblasts Xenotransplantation. 2014;21(3):291-300 Assignment of two human α-1,3-galactosyltransferase gene sequences (GGTA1 and GGTA1P) to chromosomes 9q33–q34 and 12q14–q15 Genomics. 1992;12(3):613-615 Generation ofα-1,3-Galactosyltransferase-Deficient Porcine Embryonic Fibroblasts by CRISPR/Cas9-Mediated Knock-in of a Small Mutated Sequence and a Targeted Toxin-Based Selection System Reproduction in Domestic Animals. 2015;50(5):872-880 Protection of hDAF-transgenic porcine endothelial cells against activation by human complement: role of the membrane attack complex Xenotransplantation. 2002;9(2):97-105 |
